1.          NO donor inhibits proliferation and induces apoptosis by targeting PI3K/   AKT/mTOR and MEK/ERK pathways in hepatocellular carcinoma cells, Cancer   Chemotherapy and Pharmacology, 2019, 84:1303–1314

2.          Protein phosphatase 2A activation mechanism contributes to JS-K induced

3.          caspase-dependent apoptosis in human hepatocellular carcinoma cells.   Journal of Experimental & Clinical Cancer Research, 2018,37:142.

4.          Protein phosphatase 2A mediates JS-K-induced apoptosis by affecting   Bcl-2 family proteins in human hepatocellular carcinoma HepG2 cells. Journal   of Cellular Biochemistry. 2018,119(8):6633-6643

5.          JS-K as a nitric oxide donor induces apoptosis via the ROS/Ca2+/caspase   mediated mitochondrial pathway in HepG2 cells. Biomedicine &   Pharmacotherapy. 2018, 107:1385-1392

6.          The nitric oxide prodrug JS-K induces Ca (2+) -mediated apoptosis in   human hepatocellular carcinoma HepG2 Cells. J Biochem Mol Toxicol. 2016,   30(4):192-199.

7.          NOAD, a novel nitric oxide donor, induces G2/M phase arrest and   apoptosis in human hepatocellular carcinoma Bel-7402 cells. Toxicol In Vitro.   2015, 29(7):1289-1297.

8.          NG as a novel nitric oxide donor induces apoptosis by increasing   reactive oxygen species and inhibiting mitochondrial function in MGC803   cells. Int Immunopharmacol. 2014, 23(1):27-36.

9.       偶氮鎓二醇盐衍生物JS-K 通过MEK/ERK 通路抑制肝纤维化抗大鼠肝癌, 中国药理学与毒理学杂志, 2020, 34(6):428-434