|
[1] Oyake T, Itoh K, Motohashi H, et al. Bach proteins belong to a novel family of BTB-basic leucine zipper transcription factors that interact with MafK and regulate transcription through
the NF-E2 site [J]. Mol Cell Biol, 1996, 16(11): 6083-6095.
[2] Yoshida C, Tokumasu F, Hohmura K I, et al. Long range interaction of cis-DNA elements mediated by architectural transcription factor Bach1 [J]. Genes Cells, 1999, 4(11): 643-655.
[3] Ogawa K, Sun J, Taketani S, et al. Heme mediates derepression of Maf recognition element through direct binding to transcription repressor Bach1 [J]. EMBO J, 2001, 20(11): 2835-2843.
[4] Suzuki H, Tashiro S, Hira S, et al. Heme regulates gene expression by triggering Crm1-dependent nuclear export of Bach1 [J]. EMBO J, 2004, 23(13): 2544-2553.
[5] Dhakshinamoorthy S, Jain A K, Bloom D A, et al. Bach1 competes with Nrf2 leading to negative regulation of the antioxidant response element (ARE)-mediated NAD(P)H: quinone oxidoreductase 1 gene expression and induction in response to antioxidants [J]. J Biol Chem, 2005, 280(17): 16891-16900.
[6] Lee J, Lee J, Farquhar K S, et al. Network of mutually repressive metastasis regulators can promote cell heterogeneity and metastatic transitions [J]. Proc Natl Acad Sci USA, 2014, 111(3): E364-E373.
[7] Lau A, Villeneuve N F, Sun Z, et al. Dual roles of Nrf2 in cancer [J]. Pharmacol Res, 2008, 58(5/6): 262-270.
[8] Zhuang C, Miao Z, Sheng C, et al. Updated research and applications of small molecule inhibitors of Keap1-Nrf2 protein-protein interaction: a review [J]. Curr Med Chem, 2014, 21(16): 1861-1870.
[9] Yamasaki C, Tashiro S, Nishito Y, et al. Dynamic cytoplasmic anchoring of the transcription factor Bach1 by intracellular hyaluronic acid binding protein IHABP [J]. J Bio Chem, 2005,
137(3): 287-296.
[10] Meng D, Wang X, Chang Q, et al. Arsenic promotes angiogenesis in vitro via a heme oxygenase-1-dependent mechanism [J]. Toxicol Appl Pharmacol, 2010, 244(3): 291-299.
[11] 刘俊许, 蒋丽, 魏香香, 等. 转录因子Bach1 对人微血管内皮细胞的作用研究[J]. 中国病理生理杂志, 2014(12): 2195-2200.
[12] Jiang L, Yin M, Wei X, et al. Bach1 represses wnt/beta-catenin signaling and angiogenesis [J]. Circ Res, 2015, 117(4): 364-375.
[13] Warnatz H J, Schmidt D, Manke T, et al. The BTB and CNC homology 1 (BACH1) target genes are involved in the oxidative stress response and in control of the cell cycle [J]. J Biol
Chem, 2011, 286(26): 23521-23532.
[14] Balan M, Pal S. A novel CXCR3-B chemokine receptor-induced growth-inhibitory signal in cancer cells is mediated through the regulation of Bach-1 protein and Nrf2 protein nuclear translocation [J]. J Biol Chem, 2014, 289(6): 3126-3137.
[15] Wang X, Liu J, Jiang L, et al. Bach1 induces endothelial cell apoptosis and cell-cycle arrest through ROS generation [J]. Oxidative Medicine & Cellular Longevity, 2016, 2016: 1-13.
[16] Fang M, Hutchinson L, Deng A, et al. Common BRAF(V600E)-directed pathway mediates widespread epigenetic silencing in colorectal cancer and melanoma [J]. Proc Natl Acad Sci USA, 2016, 113(5): 1250-1255.
[17] Chapple S J, Keeley T P, Mastronicola D, et al. Bach1 differentially regulates distinct Nrf2-dependent genes in human venous and coronary artery endothelial cells adapted to physiological oxygen levels [J]. Free Radic Biol Med, 2016, 92: 152-162.
[18] Omura S, Suzuki H, Toyofuku M, et al. Effects of genetic ablation of Bach1 upon smooth muscle cell proliferation and atherosclerosis after cuff injury [J]. Genes Cells, 2005, 10(3): 277-285.
[19] Yano Y, Ozono R, Oishi Y, et al. Genetic ablation of the transcription repressor Bach1 leads to myocardial protection against ischemia/reperfusion in mice [J]. Genes Cells, 2006, 11(7): 791-803.
[20] Peake B F, Nicholson C K, Lambert J P, et al. Hydrogen sulfide preconditions the db/db diabetic mouse heart against ischemia-reperfusion injury by activating Nrf2 signaling in an Erkdependent manner [J]. Am J Physiol Heart Circ Physiol, 2013, 304(9): H1215-H1224.
[21] Mito S, Ozono R, Oshima T, et al. Myocardial protection against pressure overload in mice lacking Bach1, a transcriptional repressor of heme oxygenase-1 [J]. Hypertension, 2008, 51(6): 1570-1577.
|